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Akt → Shp-2: " Shp-2 mediates v-Src induced morphological changes and activation of the anti-apoptotic protein kinase Akt "
Akt → v-Src: " Shp-2 deficiency also reduced v-Src induced activation of the anti-apoptotic protein kinase Akt "
Akt → Shp-2: " Shp-2 deficiency also reduced v-Src induced activation of the anti-apoptotic protein kinase Akt "
v-Src → p85: " Activation of PI3-kinase by v-Src may be mediated by the association of the adapter protein Cbl with the p85 subunit "
v-Src → Cbl: " Activation of PI3-kinase by v-Src may be mediated by the association of the adapter protein Cbl with the p85 subunit "
v-Src → PI3-kinase: " Activation of PI3-kinase by v-Src may be mediated by the association of the adapter protein Cbl with the p85 subunit "
PI3-kinase → p85: " Activation of PI3-kinase by v-Src may be mediated by the association of the adapter protein Cbl with the p85 subunit "
PI3-kinase → Cbl: " Activation of PI3-kinase by v-Src may be mediated by the association of the adapter protein Cbl with the p85 subunit "
Cbl ⊣ Shp-2: " Since activation of Akt is dependent on PI3-kinase, this suggests that the effect of Shp-2 on Akt activation may be mediated , at least in part, by its effects on the interaction between PI3-kinase and Cbl "
PI3-kinase ⊣ Shp-2: " Since activation of Akt is dependent on PI3-kinase, this suggests that the effect of Shp-2 on Akt activation may be mediated , at least in part, by its effects on the interaction between PI3-kinase and Cbl "
Akt → Cbl: " Since activation of Akt is dependent on PI3-kinase, this suggests that the effect of Shp-2 on Akt activation may be mediated , at least in part, by its effects on the interaction between PI3-kinase and Cbl "
Akt → PI3-kinase: " Since activation of Akt is dependent on PI3-kinase , this suggests that the effect of Shp-2 on Akt activation may be mediated, at least in part, by its effects on the interaction between PI3-kinase and Cbl "
Akt — Shp-2: " Since activation of Akt is dependent on PI3-kinase, this suggests that the effect of Shp-2 on Akt activation may be mediated, at least in part, by its effects on the interaction between PI3-kinase and Cbl "