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CA2 — CALB1

Text-mined interactions from Literome

Enomoto et al., Endocrinology 1992 (Vitamin D Deficiency) : In this study we examined the relative contributions of transcriptional and posttranscriptional mechanisms in the 1,25- ( OH ) 2D3 control of calbindin-D28K mRNA expression in primary chick kidney cells and investigated the effect of extracellular Ca2+ on calbindin-D28K gene expression in the presence and absence of hormone
Díaz de Barboza et al., Eur J Histochem 2003 (Retinal Degeneration) : A possible role of calbindin in the control of intracellular Ca2+ in neurons of this circuit when the visual transmission arrives to the optic tectum remains to be studied
Müller et al., J Neurosci 2005 (Calcium Signaling) : Numerical simulations showed that 40 microm calbindin-D28k is capable of tuning Ca2+ microdomains associated with action potentials at the mouth of single or clustered Ca2+ channels : calbindin-D28k reduces the increment in free Ca2+ at a distance of 100 and 200 nm by 20 and 35 %, respectively, and strongly accelerates the collapse of the Ca2+ gradient after cessation of Ca2+ influx ... These data suggest that calbindin-D28k equips hippocampal neurons with approximately 160 microm mobile, high-affinity Ca2+ binding sites ( kappa ( S ) approximately 200 ) that slow and reduce global Ca2+ signals while they enhance the spatiotemporal fidelity of submicroscopic Ca2+ signals
Koster et al., Cell Calcium 1995 : The role of calbindin-D28K , in transcellular Ca2+ transport and Ca2+ signaling in rabbit cortical collecting system was investigated