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CD14 — JUN

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Imai et al., Infect Immun 2000 : Since TGF-beta1 pretreatment inhibited LPS stimulated expression of c-fos and c-jun genes and also the binding of nuclear proteins to the consensus sequence of the binding site for activation protein 1 (AP-1), a heterodimer of c-Fos and c-Jun, in the cells, TGF-beta1 inhibition of CD14 expression may be a consequence of downregulation of AP-1
Iwahashi et al., J Immunol 2000 : Prostaglandin E2 stimulates AP-1 mediated CD14 expression in mouse macrophages via cyclic AMP dependent protein kinase A ... Therefore, the present study demonstrates that PGE2 has the ability to stimulate AP-1 mediated expression of CD14 in mouse macrophages via cAMP dependent protein kinase A
Welters et al., Eur J Anaesthesiol 2007 : The decrease in CD14 expression caused by morphine may play a role in inhibition of activator protein 1 activation following lipopolysaccharide treatment of phagocytes
Nareika et al., J Endocrinol 2008 : Further investigations using transcription factor activity assays and gel shift assays revealed that high glucose augmented LPS stimulated CD14 expression by enhancing transcription factor nuclear factor kappaB (NFkappaB) and activator protein-1 (AP-1) activities