◀ Back to RELA

CD38 — RELA

Text-mined interactions from Literome

Kaku et al., Int Immunol 2002 : Activation of NF-kappaB-like proteins by CD38 ligation is not observed in splenic B cells from Bruton's tyrosine kinase (Btk)-deficient ( Btk ( -/- ) ) mice, with inhibitors of protein kinase C ( PKC ) and phosphatidylinositol (PI)-3 kinase also suppressing NF-kappaB activation in CD38 activated B cells
Fedele et al., Eur J Immunol 2004 : De novo synthesized CD38 is enzymatically active, and its expression in mature ( m ) MDDC is dependent on NF-kappa B activity
Shah et al., Eur J Cancer Prev 2006 (Colorectal Neoplasms) : Chronic c-9,t-11-CLA and t-10 , c-12-CLA treatment inhibited DCA induced PKC beta1 and PKC delta translocation and also inhibited NF-kappaB activation
Kang et al., FASEB J 2006 : The results indicate that TNF-alpha induced CD38 expression involves NF-kappaB expression and its activation and dexamethasone inhibits CD38 expression through NF-kappaB dependent and -independent mechanisms
Manjarrez-Orduño et al., Eur J Immunol 2007 : The cross linking of the surface molecule CD38 induces calcium mobilization, protein phosphorylation and NF-kappaB translocation into the nucleus, ultimately leading to proliferation and isotype switching toward IgG1
Tirumurugaan et al., Am J Physiol Lung Cell Mol Physiol 2007 (MAP Kinase Signaling System) : These results indicate that regulation of CD38 expression through p38 and JNK MAPKs involves NF-kappaB and AP-1 activation, and ERK and p38 MAPKs also regulate expression posttranscriptionally through message stability
Abraham et al., Virus Res 2008 : Further, induction of H-2Q4 and H-2T23 by JEV was independent of NF-kappaB but type 1 IFN dependent while H-2T10 was dependent on NF-kappaB and type 1 IFN independent
Tirumurugaan et al., Respir Res 2008 (Bronchial Hyperreactivity) : Regulation of CD38 expression in HASM cells involves the transcription factor NF-kappaB , and glucocorticoids inhibit this expression through NF-kappaB dependent and -independent mechanisms ... The EMSA results and the cd38 promoter-reporter assays confirm the functional role of NF-kappaB , AP-1 and GREs in the cd38 promoter in the transcriptional regulation of CD38
Kang et al., Am J Physiol Lung Cell Mol Physiol 2008 (MAP Kinase Signaling System) : The transcriptional and posttranscriptional regulation of CD38 expression involves signaling through MAPKs and requires activation of NF-kappaB and activator protein-1 (AP-1)