Gene interactions and pathways from curated databases and text-mining

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CD5 — IL6

Text-mined interactions from Literome

Gibson et al., J Mol Cell Immunol 1990 : Data presented here and elsewhere suggest that this CD5+ B-cell inducing activity is not mediated by IL-1, IL-2, IL-3, IL-4, IL-5, IL-6 , IFN-gamma, GM-CSF, or TNF
Masuda et al., Leuk Res 1991 (Leukemia-Lymphoma, Adult T-Cell) : However, CD5+ or CD1+ acute leukemia cells were not stimulated by IL-3 or IL-6
Defrance et al., Eur J Immunol 1989 (Leukemia, Lymphocytic, Chronic, B-Cell) : Recombinant IL 1 alpha, IL 2, IL 3, IL 5, IL 6 , tumor necrosis factor (TNF)-alpha, IFN-gamma and granulocyte macrophage colony stimulating factor neither inhibit nor stimulate CD5 expression on B cells, therefore suggesting that the capacity to down-regulate CD5 expression on B cells is specific to IL 4
Kanno et al., Autoimmunity 1993 (Lupus Erythematosus, Systemic) : However, significant amounts of IgG antibodies were also produced, in the presence of IL-6 , by CD5+Lp-3+sIgM+ , but not by CD5-Lp-3+sIgM+ B cells from 6-month-old mice
Spencer et al., Int Immunol 1997 : A role for IL-12 in the aging process was suggested when it was found that recombinant IL-12 ( rIL-12 ) directly stimulated splenic CD5+ B cells to secrete IL-10, and both CD5+ and CD5- B cells could be directly induced to produce IL-6 in response to rIL-12