◀ Back to CA2

CA2 — KCNMA1

Text-mined interactions from Literome

Yamamura et al., Br J Pharmacol 2001 : BK channel activation by NS-1619 is partially mediated by intracellular Ca2+ release in smooth muscle cells of porcine coronary artery
Chung et al., Expert Opin Investig Drugs 2002 (Cough) : Local anaesthetics, blockers of sodium dependent channels and maxi-K Ca2+ dependent channel activators of afferent nerves are inhibitors of the cough reflex
Chung et al., Pulm Pharmacol Ther 2002 (Cough) : Local anaesthetics, blockers of sodium dependent channels, and maxi-K CA2+ dependent channel activators of afferent nerves are inhibitors of the cough reflex
Liu et al., EMBO J 2004 : Assembly of a Ca2+ dependent BK channel signaling complex by binding to beta2 adrenergic receptor
Lu et al., J Physiol 2006 (Calcium Signaling...) : As MaxiK senses and regulates membrane voltage and intracellular Ca2+ , it links cell excitability with cell signalling and metabolism
Misonou et al., J Comp Neurol 2006 : Immunolocalization of the Ca2+ activated K+ channel Slo1 in axons and nerve terminals of mammalian brain and cultured neurons
Chiang et al., J Exp Biol 2006 : In the Rhabditidae family, although M4 normally has no effect on the terminal bulb, we found that M4 can stimulate the terminal bulb in C. elegans if the Ca2+ activated K+ channel SLO-1 is inactivated
Latorre et al., Biol Res 2006 (Ion Channel Gating) : The BK channel is activated by internal Ca2+ , and using chimeric channels and mutagenesis, three distinct Ca2+ dependent regulatory mechanisms with different divalent cation selectivity have been identified in its large COOH-terminus
Kaufmann et al., J Comp Neurol 2009 : Such increases in cytosolic [Ca2+ ] may be sufficient for BK channel activation
Latorre et al., J Physiol 2010 : Allosteric interactions and the modular nature of the voltage- and Ca2+ activated ( BK) channel
Guggino et al., Am J Physiol 1987 : We conclude that cultures of MTAL cells GRB-MAL1 retain at least two of the properties of the mature phenotype, namely, an apical K+ conductance and a sensitivity to loop diuretics ; the most frequently occurring channel in the apical cell membrane is a Ca2+ activated , maxi-K+ channel ; and, finally Ca2+ activated K+ channels may play a role in generating the apical K+ conductance in cultured MTAL cells
Taniguchi et al., Am J Physiol 1989 : The effects of membrane stretch on Ca2+ activated ( maxi) K+ channels were examined in the apical membrane of cultured medullary thick ascending limb ( MTAL ) cells
Romero et al., Am J Physiol 1998 : Evidences that activation of the maxi-K+ channel by ANG II requires a rise in intracellular Ca2+ concentration ([Ca2+]i) as an intermediate step were the shift of the open probability of the channel-membrane potential relationship to less positive membrane potentials and the sustained increase in [ Ca2+ ] i in fura 2-loaded myocytes