◀ Back to IL1B

IL1B — PRKAA2

Text-mined interactions from Literome

Giri et al., J Neurosci 2004 (Inflammation) : A pharmacological activator of AMPK , 5-amino-4-imidazole carboxamide riboside ( AICAR ) inhibited lipopolysaccharide (LPS) induced expression of proinflammatory cytokines ( tumor necrosis factor alpha, interleukin-1beta , and interleukin-6 ) and inducible nitric oxide synthase in primary rat astrocytes, microglia, and peritoneal macrophages
Ropelle et al., Endocrinology 2007 (Anorexia...) : In parallel, the pharmacological activation of hypothalamic AMPK in TB animals markedly reduced the hypothalamic production of inducible nitric oxide synthase, IL-1beta , and TNF-alpha and modulated the expression of proopiomelanocortin, a hypothalamic neuropeptide that is involved in the control of energy homeostasis
Łabuzek et al., Neurotoxicology 2010 : The presented evidence supports the conclusion that AMPK activated by AICAR is involved in regulation of ROS and cytokine production ( IL-1 beta , TNF-alpha ( 6h ), IL-10 and TGF-beta ) as well as arginase I and PGC-1alpha expression