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PRNP — TNF

Text-mined interactions from Literome

Fabrizi et al., J Biol Chem 2001 : PrP- ( 106 -- 126 ) caused the release of TNF-alpha as detected by enzyme linked immunosorbent assay, and a blocking antibody, anti-TNF-alpha, abolished NOS-II induction elicited by this peptide
Veerhuis et al., Brain Res 2002 (Prion Diseases) : Since microglial COX-2 expression and PGE ( 2 ) synthesis are increased in human and experimental prion diseases, we investigated the effects of the NSAIDs indomethacin and BF389, an experimental COX-2 selective inhibitor, on the PrP105-132 induced microglial IL-6 and TNF-alpha synthesis in vitro
Sauer et al., Free Radic Biol Med 2003 : Regulation of intrinsic prion protein by growth factors and TNF-alpha : the role of intracellular reactive oxygen species
Yang et al., Biochem J 2005 : TNFalpha induces the expression and recombinant promoter activities of GCLC, GCLM and GSS in H4IIE cells
Pradines et al., J Neurochem 2009 (Second Messenger Systems) : We established that in 1C11 derived neuronal cells antibody mediated PrP ( C ) ligation triggered tumor necrosis factor (TNF)-alpha release, through recruitment of the metalloproteinase TNF-alpha converting enzyme (TACE)
Scalabrino et al., Exp Neurol 2012 (Disease Models, Animal...) : However, as it is known that Cbl deficiency damages myelin by increasing tumor necrosis factor (TNF)-a and decreasing epidermal growth factor (EGF) levels in rat spinal cord ( SC ), and that TNF-a and EGF regulate PrP ( C ) expression in vitro, we investigated whether Cbl deficiency modifies SC PrP ( C ) and PrP ( C ) -mRNA levels in Cbl-D rats
Kordek et al., Proc Natl Acad Sci U S A 1996 (Creutzfeldt-Jakob Syndrome) : To clarify the role of TNF-alpha in experimental CJD , we investigated the expression of TNF-alpha in brain tissues from CJD virus infected mice at weekly intervals after inoculation by reverse transcription coupled PCR, Northern and Western blot analyses, and immunocytochemical staining