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MAPK15 — RENBP

Text-mined interactions from Literome

Zill et al., Biochem Biophys Res Commun 2003 : RAGE mediated MAPK activation by food derived AGE and non-AGE products
Ouyang et al., Sheng Li Xue Bao 2003 (Atherosclerosis...) : The p44/42 MAPK activity was significantly enhanced by AGE
Chang et al., Kidney Int 2004 (MAP Kinase Signaling System) : Activation of PPAR-gamma by rosiglitazone inhibited AGE induced iNOS expression, nitrite release, and p38 MAPK activation in mesangial cells
Li et al., Xi Bao Yu Fen Zi Mian Yi Xue Za Zhi 2003 : High glucose, AGE , high insulin and H ( 2 ) O ( 2 ) could activate P38MAPK by oneself, and increased notably expression of phosphorylated-P38MAPK and P-selectin on cell line ECV304 ; Expression of P-selectin was inhibited significantly by SB203580
Lu et al., Proc Natl Acad Sci U S A 2004 (Inflammation) : However, AGE stimulated NF-kappaB activity and mitogen activated protein kinase ( MAPK ) ( p44/42 ) phosphorylation were found markedly suppressed in R1-MC. Additionally, AGE stimulated macrophage chemotaxis protein 1 and RAGE overexpression were abolished in R1-MC
Li et al., Life Sci 2007 : This study was designed to evaluate the effect of AGE on accumulation of reactive oxygen species ( ROS ), apoptosis, mitogen activated protein kinase ( MAPK ) activation and nuclear O-GlcNAcylation in fetal human cardiac myocytes ... Collectively, these results indicated that AGE or its precursor MG may trigger ROS generation, apoptosis, MAPK activation and nuclear O-GlcNAcylation in human cardiac myocytes, in a manner reminiscent of high extracellular glucose
Huang et al., Toxicological sciences : an official journal of the Society of Toxicology 2009 (Hypertrophy) : Moreover, AGE induced extracellular signal regulated kinase/c-Jun N-terminal kinase/p38 mitogen activated protein kinase activation was markedly blocked by antireceptor for AGE ( RAGE ), SNAP, SNP, NAC, and taurine
Liu et al., FEBS J 2009 : Similar to lipopolysaccharide, AGE-HSA stimulation induced mitogen activated protein kinase phosphorylation and nuclear factor-kappaB nuclear translocation in human umbilical vein endothelial cells, which was further enhanced by a combination of the two stimulants
Wang et al., BMC cell biology 2011 : AGE-BSA down-regulated Cx43 expression in HAEC, mainly through reduced Cx43 transcription, and the process involved activation of ERK and p38 MAPK
Shi et al., PloS one 2013 (Corneal Diseases...) : We also found that AGE-BSA induced JNK and p38 MAPK phosphorylation ... In addition, NAC completely blocked phosphorylation of JNK and p38 MAPK induced by AGE-BSA