Gene interactions and pathways from curated databases and text-mining

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IL6 — SELE

Text-mined interactions from Literome

Cockerill et al., Circulation 2001 (Acute Disease...) : Elevation of plasma high-density lipoprotein concentration reduces interleukin-1 induced expression of E-selectin in an in vivo model of acute inflammation
Landis et al., Arthritis Rheum 2002 : Human peripheral blood monocytes were differentiated for 1-7 days in vitro and examined with respect to 1 ) uptake of MSU crystals, 2 ) expression of macrophage, dendritic cell, and activation markers, 3 ) secretion of tumor necrosis factor alpha (TNFalpha), interleukin 1beta (IL-1beta), IL-6 , and IL-10, 4 ) activation of endothelial E-selectin expression, and 5 ) enhancement of secondary neutrophil recruitment by endothelial cells ... MSU crystals induced TNFalpha, IL-1beta, and IL-6 ( but not IL-10 ) secretion in undifferentiated monocytes, which in turn promoted endothelial cell E-selectin expression and secondary neutrophil capture under shear flow
Leeuwenberg et al., J Immunol 1990 : Neither IFN-beta nor IL-6 had any effect on the expression of ELAM-1
Leeuwenberg et al., Eur J Immunol 1991 (Leukemia, Myeloid) : ELAM-1 expression can selectively be induced on human umbilical vein endothelial cells ( HUVEC ) by tumor necrosis factor, interleukin 1 and lipopolysaccharide
Smadja et al., Pediatr Cardiol 2010 (Heart Defects, Congenital...) : The study investigated soluble markers of endothelial damage or activation ( thrombomodulin, soluble endothelial protein C receptor, and soluble E-selectin ), inflammation ( interleukin-6 ), and angiogenic cytokine levels [ vascular endothelial growth factor ( VEGF ) and placental growth factor (PlGF) ] in 26 patients with CHD, 16 with reversible PAH ( median age, 2 years ) and 10 with irreversible PAH ( median age, 9 years )
Lewis et al., Mol Cell Biol 1994 : In addition, an essential role for cooperative interaction between the two NF-kappa B complexes is shown by the requirement for both NF-kappa B sites to mediate E-selectin promoter activation by interleukin-1 and p50/p65 expression
Weston et al., Transpl Immunol 1995 (Kidney Failure, Chronic) : E-Selectin is a 115-kDa cell surface glycoprotein transiently expressed on vascular endothelium in response to interleukin-1 and tumour necrosis factor-alpha with a peak in expression at four hours
Del Papa et al., Arthritis Rheum 1996 (Wegener Granulomatosis) : We found that AECA IgG from WG patients do not display any significant cytotoxicity but are able to up-regulate the expression of E-selectin , intercellular adhesion molecule 1 and vascular cell adhesion molecule 1 and to induce the secretion of IL-1 beta, IL-6 , IL-8, and MCP-1
Lou et al., J Neuroimmunol 1997 : TBP I completely abolished TNF induced IL-6 production and E-selectin induction , while it partially inhibited TNF induced IL-8 production and up-regulation of ICAM-1 and VCAM-1
Ray et al., Biochem J 1997 : Induction of the E-selectin promoter by interleukin 1 and tumour necrosis factor alpha, and inhibition by glucocorticoids
Miller et al., J Leukoc Biol 1998 : Utilizing neutralizing antibodies, we show that CD40L mediated tissue factor and thrombomodulin modulation, as well as E-selectin and VCAM-1 upregulation, is independent of tumor necrosis factor alpha, interleukin-1alpha , or interleukin-1beta production
Friedrichs et al., Arterioscler Thromb Vasc Biol 1998 : Inhibition of tumor necrosis factor-alpha- and interleukin-1 induced endothelial E-selectin expression by thiol modifying agents