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EPHB2 — SOD1

Text-mined interactions from Literome

Cho et al., Biochem Biophys Res Commun 1999 : However, SOD enhanced the silica induced ERK phosphorylation, indicating a role for H ( 2 ) O ( 2 ) in ERK activation
Noshita et al., J Neurosci 2002 (Ischemic Attack, Transient) : These results suggest that phosphorylation of ERK1/2 may be involved in apoptosis or cell death after transient FCI and that SOD1 may attenuate apoptotic cell death mediated by the mitogen activated protein kinase/ERK pathway
Kulich et al., J Biosci 2003 : However, exogenously added SOD1 and heat inactivated catalase had no effect on either toxicity or sustained ERK phosphorylation
Conde de la Rosa et al., J Hepatol 2006 (MAP Kinase Signaling System...) : ( 3 ) PEG-SOD prevents ERK1/2- , JNK- phosphorylation, caspase activation and apoptosis induced by menadione
Lin et al., Atherosclerosis 2007 (MAP Kinase Signaling System) : Phosphorylation studies on ERK1/2, JNK, and p38, three major subgroups of mitogen activator protein kinases, demonstrated that Cu, Zn-SOD and/or catalase overexpression suppressed ERK1/2 and JNK phosphorylation
Borrás et al., FASEB J 2006 : Phosphorylation of extracellular regulated kinase (ERK)1/2 led to an activation of NFkappaB, as indicated by increased p50 subunit expression in nuclear extracts, and increased mRNA levels of the antioxidant enzyme manganese-superoxide dismutase ( MnSOD )
Shin et al., Free Radic Biol Med 2008 : A pretreatment with NAC or catalase, but not SOD , attenuated the phosphorylation of ERK , JNK, and p38 kinase by heat shock
Juarez et al., Proc Natl Acad Sci U S A 2008 (Neovascularization, Pathologic) : Pretreatment with exogenous H ( 2 ) O ( 2 ) or with the phosphatase inhibitor vanadate abrogates the inhibition of ERK1/2 phosphorylation induced by ATN-224 or SOD1 siRNA treatments
Secondo et al., Biochem Biophys Res Commun 2008 : The Cu-Zn superoxide dismutase ( SOD1 ) inhibits ERK phosphorylation by muscarinic receptor modulation in rat pituitary GH3 cells
Cassano et al., J Biol Chem 2010 (MAP Kinase Signaling System) : Long term activation of ERK1/2 by NGF requires SOD activation, low levels of hydrogen peroxide, and the integrity of the microtubular cytoskeleton ... Local activation of ERK1/2 by Ki-Ras stimulates mitochondrial SOD , which reduces reactive oxygen species and produces H ( 2 ) O ( 2 )
Akool et al., Biochem Pharmacol 2012 (MAP Kinase Signaling System) : The MEK inhibitor U0126, the reactive oxygen species ( ROS ) scavenger N-acetyl-cysteine (NAC), a cell-permeant superoxide dismutase ( SOD ) and stigmatellin, an inhibitor of mitochondrial cytochrome bc1 complex strongly attenuated the increase in ERK1/2 phosphorylation triggered by PPIase inhibitors
Armstead et al., J Neurotrauma 2012 (Brain Injuries) : ERK upregulation was blocked by BQ-123 and PEG-SOD