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CCL4 — JUN

Pathways - manually collected, often from reviews:

• BioCarta pertussis toxin-insensitive ccr5 signaling in macrophage: AP-1 complex (FOS-JUN) → MIP-1beta (CCL4) (transcription, activates)
• KEGG Toll-like receptor signaling pathway: FOS/JUN → CCL4 (protein-protein, expression)

Text-mined interactions from Literome

Li et al., J Clin Endocrinol Metab 2011 (Inflammation...) : In IL-1ß- or TNF-a stimulated first trimester decidual cells, NF?B inhibitor suppressed production of all six chemokines ; JUN NH2-terminal kinase inhibitor inhibited secretion of CCL2, CCL4 , and CCL5 ; and MAPK kinase and p38 inhibitor decreased production of CXCL8
Tsai et al., J Virol 2013 : Furthermore, EBV latent membrane protein 1 (LMP1) mediated Jun N-terminal protein kinase activation was responsible for upregulation of CCL3 and CCL4