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FOXM1 — PCNA

Text-mined interactions from Literome

Major et al., Mol Cell Biol 2004 (MAP Kinase Signaling System) : We demonstrated that FoxM1B transcriptional activity requires binding of either S-phase or M-phase Cdk-cyclin complexes to mediate efficient Cdk phosphorylation of the FoxM1B Thr 596 residue, which is essential for recruitment of p300/CBP coactivator proteins
Santin et al., Virology 2005 (Uterine Cervical Neoplasms) : Cyclin dependent kinase inhibitor 2A ( CDKN2A/p16 ), mesoderm-specific transcript, forkhead box M1 , v-myb myeloblastosis viral oncogene homolog ( avian ) -like2 ( v-Myb ), minichromosome maintenance proteins 2, 4, and 5, cyclin B1, prostaglandin E synthase (PTGES), topoisomerase II alpha (TOP2A), ubiquitin conjugating enzyme E2C, CD97 antigen, E2F transcription factor 1, and dUTP pyrophosphatase were among the most highly overexpressed genes in CVX when compared to NCK
Laoukili et al., Mol Cell Biol 2008 : We found that cyclin A/cdk complexes are required to phosphorylate and activate FoxM1 during G ( 2 ) phase
Dai et al., Cancer Res 2010 (Brain Neoplasms...) : Furthermore, overexpression of FoxM1B in immortalized NHAs increased the expression of survivin, cyclin D1, and cyclin E, which are important molecules for tumor growth