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S100A4 — SLC22A3

Text-mined interactions from Literome

Amatangelo et al., Carcinogenesis 2012 (Prostatic Neoplasms) : Characteristically, EMT involved adoption of a spindle shaped morphology, loss of E-cadherin and increased expression of Vimentin, Fibronectin and Fibroblast Specific Protein-1 ( S100A4 )
Zheng et al., Oncol Rep 2013 (Carcinoma, Hepatocellular...) : Ectopic expression of metastasin in vitro induced typical epithelial-mesenchymal transition (EMT) in Hep3B cells including higher capacity of both migration and invasion, increased expression of both Vimentin and N-cadherin and decreased expression of E-cadherin ... Collectively, the present results suggest that metastasin leads to HCC EMT partly through upregulating SNAI1 and contributes to poor prognosis following radical liver resection