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CTF1 — JAK1

Text-mined interactions from Literome

Hamanaka et al., Circ Res 2001 (Hypotension) : These results suggest that JAB and CIS3 serve as endogenous inhibitors of CT-1 mediated JAK-STAT signaling in the cardiovascular system in vivo
Freed et al., Mol Cell Biochem 2003 (Disease Models, Animal...) : CT-1 induced rapid phosphorylation of Jak , Jak2, STAT1, STAT3, p42/44 MAPK and Akt in cultured adult cardiac fibroblasts
Wu et al., Mol Cell Biochem 2006 (Cardiomegaly) : Simvastatin was proved, in a dose independent manner, to decrease cardiacmyocytes size as well as protein synthesis, and inhibit ANP mRNA synthesis and JAK-STAT protein expression induced by CT-1 in cardiacmyocytes.Conclusion : These results suggest that simvastatin can ameliorate cardiacmyocytes hypertrophyin vitrovia JAK-STAT signaling pathways