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SOAT1 — STAT1

Text-mined interactions from Literome

Liu et al., Zhonghua Yi Xue Za Zhi 2001 : STAT1 and STAT3 antisense oligonucleotides inhibited both STAT DNA binding activity and TIMP1 mRNA expression in mesangial cells
Chen et al., Kidney Int 2002 : EMSA showed that STAT1 and STAT3 antisense oligonucleotides could inhibit both thrombin induced STAT-DNA binding activities and TIMP-1 mRNA expression ; the supershift assay showed that the SIF band consisted of STAT1 and STAT3 proteins
Parisien et al., J Virol 2002 : Selective STAT protein degradation induced by paramyxoviruses requires both STAT1 and STAT2 but is independent of alpha/beta interferon signal transduction
Velichko et al., J Biol Chem 2002 : The role of STAT1 and STAT2 in IFN dependent JAK-STAT signaling is well established ; however, the role of STAT3 and its activation by IFNs remains unclear
Bovolenta et al., J Immunol 2002 (HIV Infections...) : The emergence of STAT activation , namely of STAT1 , in culture was mostly related to the secretion of IFN-gamma
Precious et al., J Gen Virol 2005 : As previously reported, STAT1 could not be detected in human cells that expressed SV5 V protein constitutively, whilst STAT2 could not be detected in human cells that expressed hPIV2 V protein, although the levels of STAT1 may also have been reduced in some human cells infected with hPIV2
Lin et al., J Virol 2006 : We propose a model by which the binding of HCV core to STAT1 results in decreased P-STAT , blocked STAT1 heterodimerization to STAT2, and, therefore, reduced IFN stimulated gene factor-3 binding to DNA and disrupted IFN stimulated gene transcription
Shang et al., Cancer Sci 2007 (Carcinoma, Renal Cell...) : The results suggest that the resistance of RCC to IFN-alpha is associated with the lack of Jak1, Tyk2 and Stat1 expression and defective Jak-Stat activation , but not with a lack of IFN-alpha receptor, suppressors of cytokine signaling induction or other factors examined
Hazari et al., Virology journal 2007 : All the cell lines show defect in the JAK-STAT signaling and phosphorylation of STAT 1 and STAT 2 proteins were strongly inhibited due to reduced expression of Tyk2 and Jak-1 protein
Krejci et al., J Cell Sci 2008 : Moreover, addition of active STAT1 and STAT3 to the FGF signal, by means of cytokine treatment, SRC mediated STAT activation or expression of constitutively active STAT mutants does not sensitize RCS chondrocytes to FGF mediated growth arrest
Hu et al., J Leukoc Biol 2008 : In this study, we analyzed the regulation of STAT signaling during maturation of human DCs by TNF-alpha and PGE2, which induced maturation of human DCs comparably with LPS but did not induce detectable IFN-beta production or Stat1 tyrosine phosphorylation
Parafioriti et al., J Orthop Sci 2009 (Achondroplasia) : Chondrocytes in cartilage biopsies of ACH children were characterized by the presence of growth arrest mediated by STAT activation ( both STAT1 and STAT5 ) and increased expression of p21 and cyclin D1, whereas no expression of either p53 or cyclin D3 could be detected