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APOB — C9ORF3

Text-mined interactions from Literome

Vohl et al., Atherosclerosis 2002 (Hyperlipoproteinemia Type II) : Multiple regression analyses showed that there was a significant association between the apo E polymorphism and LDL-cholesterol response to simvastatin only among heterozygotes for a receptor negative mutation
Keidar et al., Metabolism 1992 (Liver Neoplasms, Experimental) : Gradual fragmentation of VLDL apolipoproteins ( apo ) was noted, with apo B-100 being the first to be fragmented, followed by apo E and apo C. Degradation of Ox-VLDL by mouse peritoneal macrophages ( MPM ) was increased almost twofold in comparison to N-VLDL
Shireman et al., Proc Natl Acad Sci U S A 1977 : Unlabeled LDL and apo B-albumin complex both competitively inhibited the binding of ( 125 ) I-labeled apo B and ( 125 ) I-labeled LDL to fibroblasts
Goswami et al., Clin Biochem 2012 (Coronary Artery Disease...) : Apolipoprotein-A ( Apo-AI ) and Apolipoprotein-B (Apo-B) were estimated and small dense LDL was derived mathematically
Soutar et al., Atherosclerosis 1979 (Hypercholesterolemia) : These findings provide no support for the hypothesis that apo B synthesis is controlled by the plasma LDL
Inagaki et al., Atherosclerosis 1990 (Hypertriglyceridemia) : In spite of this, LDL cholesterol levels did not decrease, and apo B levels increased ( 98 +/- 28 to 111 +/- 27, P less than 0.05 )
Wernette-Hammond et al., Arteriosclerosis 1989 (Disease Models, Animal...) : These results indicate that the altered LDL receptors expressed on cultured WHHL rabbit cells are sufficient for apo E-dependent, receptor mediated uptake of lipoproteins
Eisenberg et al., Arteriosclerosis 1988 : Exogenous apo E-3 caused a many-fold enhancement of the metabolism of the three VLDL fractions, but LDL was not affected
Hui et al., J Biol Chem 1981 : The binding of the 125I-LDL to the liver membranes from the adult cholestyramine treated dogs or from the puppies appeared to be mediated by apo-B , E receptors which resembled the LDL receptor of human skin fibroblasts
Bradley et al., J Biol Chem 1984 (Hyperlipoproteinemias) : We conclude that apo-E of the thrombin-accessible conformation mediates uptake of HTG-VLDL1 and HTG-VLDL2 but that apo-B alone is sufficient to mediate receptor binding of IDL and LDL ; the switch from apo-E to apo-B as the primary or sufficient binding determinant occurs within the VLDL3 ( Sf 20-60 ) region of the metabolic cascade, where receptor binding first appears in VLDL subclasses from normal subjects
Gomez-Jimenez et al., Ann Biol Clin (Paris) 1995 (Diabetes Mellitus, Type 2) : On admission, diabetic subjects had significantly higher plasma levels of triglycerides and lower levels of HDL cholesterol ; during hospitalization, LDL , HDL cholesterol and apo A1 levels increased significantly
Loirdighi et al., J Cell Biochem 1997 : In parallel, [ 35S ] -methionine pulse labeling of jejunal explants revealed the concomitant inhibitory effect of hydrocortisone on apo B-100 synthesis and hydrocortisone 's stimulatory effect on apo B-48 and apo A-1
Paragh et al., Metabolism 1998 (Diabetes Mellitus, Type 2...) : The LDL induced inhibition of cholesterol synthesis and the acLDL transmitted apo E secretion were also found to be decreased in the MDMs of patients with NIDDM as compared with the obese and non-obese control groups