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EPHB2 — INPP5D

Text-mined interactions from Literome

An et al., Blood 2005 (Inflammation) : Neither disrupting phosphatase activity nor using the PI-3K pathway inhibitor LY294002 or wortmannin could significantly block SHIP1 mediated inhibition of LPS induced ERK1/2 , p38, and JNK activation and TNF-alpha production, demonstrating that SHIP1 inhibits LPS induced activation of MAPKs and cytokine production primarily by a phosphatase activity- and PI-3K independent mechanism
Trotta et al., Blood 2013 (Lymphoma...) : The enhanced NK-cell survival, expansion, activation, and tumor control that result from overexpression of miR-155 in NK cells could be explained, in part, via diminished expression of the inositol phosphatase SHIP1 and increased activation of ERK and AKT kinases