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CD19 — IRF6

Text-mined interactions from Literome

Yazawa et al., Blood 2003 : LPS induced tyrosine phosphorylation of CD19 , which was RP105 dependent but TLR4 independent
Khan et al., J Leukoc Biol 2011 : UCB ( =25 µM ) was toxic to unfractionated splenocytes, splenic T cells, B cells, macrophages, LPS stimulated CD19 ( + ) B cells, human PBMCs, and RBCs. Purified UCB also was found to be toxic to splenocytes and human PBMCs. UCB induced necrosis and apoptosis in splenocytes
Zhang et al., PloS one 2013 : In vitro treatment with BE resulted in a higher increase in the level of apoptosis in Con A-stimulated murine splenocytes, Con A-stimulated CD3 ( + ) splenocytes, lipopolysaccharide (LPS) stimulated CD19 ( + ) splenocytes, and phorbol 12-myristate 13-acetate/ionomycin stimulated Jurkat T cells, compared with that in unstimulated naïve ones ... BE treatment also led to a loss of mitochondrial membrane potential, an increase of cytochrome c release from mitochondria to the cytosol, a decrease in the ratio of Bcl-2/Bax, and activation of caspase-9,-3 in Con A-stimulated CD3 ( + ) splenocytes and LPS stimulated CD19 ( + ) splenocytes, while showing no impact on Fas/FasL expressions and caspase-8 activation