◀ Back to EGF

EGF — MUC1

Text-mined interactions from Literome

Ichikawa et al., Life Sci 2000 : The EGF induced increase in [ 3H ] -labeled mucin in the full-thickness mucosa was not suppressed by either NG-nitro-L-arginine ( 10 ( -5 ) M ) or 2- ( 4-carboxyphenyl ) -4,4,5,5-tetramethylimidazoline-1-oxyl-3-oxide ( 10 ( -5 ) M )
Rokutan et al., J Gastroenterol 2000 : Thus, the present experiments suggests that GGA, as well as EGF , stimulates mucin synthesis at least in part through an NO-dependent pathway, leading to an increase in the integrity of the gastric mucosa
Takeyama et al., Am J Physiol Lung Cell Mol Physiol 2001 (Lung Diseases, Obstructive) : Activation of epidermal growth factor receptors is responsible for mucin synthesis induced by cigarette smoke ... Here we show that activation of epidermal growth factor receptors (EGFR) is responsible for mucin production after inhalation of cigarette smoke in airways in vitro and in vivo
Song et al., Korean J Intern Med 2001 : The production of mucin was regulated by epidermal growth factor (EGF) in vitro
Chung et al., Eur Respir J Suppl 2001 (Inflammation...) : TGFbeta and EGF activate proliferation of fibroblasts, while activation of the EGF receptor leads to mucin gene expression
Hattrup et al., Biochem Biophys Res Commun 2004 (Breast Neoplasms...) : These studies also indicate that the association of MUC1 and APC may be increased by epidermal growth factor stimulation
Kitazaki et al., Lung Cancer 2005 (Adenocarcinoma...) : In addition, epidermal growth factor regulates mucin secretion in normal airway goblet cells
Chung et al., Curr Drug Targets 2006 (Pulmonary Disease, Chronic Obstructive) : TGFbeta and EGF activate proliferation of fibroblasts, while activation of the EGF receptor leads to mucin gene expression
Chung et al., Curr Drug Targets Inflamm Allergy 2005 (Pulmonary Disease, Chronic Obstructive) : TGFbeta and EGF activate proliferation of fibroblasts, while activation of the EGF receptor leads to mucin gene expression
Ramasamy et al., Mol Cell 2007 : The significance of the MUC1-C-galectin-3 interaction is supported by the demonstration that galectin-3 forms a bridge between MUC1 and the epidermal growth factor receptor (EGFR) and that galectin-3 is essential for EGF mediated interactions between MUC1 and EGFR
Heo et al., Phytother Res 2009 : Genistein and curcumin suppress epidermal growth factor induced MUC5AC mucin production and gene expression from human airway epithelial cells ... This study investigated whether genistein and curcumin affect epidermal growth factor (EGF) induced MUC5AC mucin production and gene expression from human airway epithelial cells
Lee et al., Phytomedicine 2011 : Glycyrrhizin and carbenoxolone were found to inhibit the production of MUC5AC mucin protein induced by EGF or PMA, and both compounds also inhibited the expression of MUC5AC mucin gene induced by EGF or PMA
Cho et al., Phytother Res 2011 : suppress epidermal growth factor- and phorbol ester induced MUC5AC mucin production and gene expression from human airway epithelial cells ... Oleanolic acid and ursolic acid were found to inhibit the production of MUC5AC mucin protein induced by EGF and PMA, and both compounds also inhibited the expression of MUC5AC mucin gene induced by EGF and PMA
Xiao et al., Invest Ophthalmol Vis Sci 2012 (Disease Models, Animal...) : In addition, EGF significantly increased the goblet cells number and MUC1 expression in the epithelium
Kim et al., Phytother Res 2012 : Phorbol ester or epidermal growth-factor induced MUC5AC mucin gene expression and production from airway epithelial cells are inhibited by apigenin and wogonin
Jin et al., Oncogene 2013 : These findings indicate that EGF induced MUC1-C expression is mediated by the PI3K?AKT pathway and the eIF4A RNA helicase, and that this response promotes EGFR signaling in an autoinductive loop
Hodges et al., Exp Eye Res 2012 : Inhibitors against the EGFR and ERK 1/2 blocked EGF stimulated mucin secretion