Verrecchia et al., J Biol Chem 2003
:
We demonstrate that, in a cellular context devoid of JNK activity ( i.e. jnk ( -/- ) fibroblasts ), interleukin-1 and tumor necrosis factor-alpha (TNF-alpha) did not inhibit the formation of SMAD-DNA complexes and the resulting SMAD-driven transcription in response to TGF-beta
Warstat et al., Ann Rheum Dis 2010
(Arthritis, Rheumatoid...) :
This IL16 regulation involved p38MAPK, ERK1/2 and SMAD2 signalling, but not NFkappaB