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IL3 — LTA

Text-mined interactions from Literome

Morath et al., Infect Immun 2002 : After purification, the commercial LTA from Bacillus subtilis and S. pyogenes but not LTA from S. aureus induced the release of tumor necrosis factor alpha, interleukin 1 beta (IL-1beta), IL-6, and IL-10 in human blood
Walker et al., J Immunol 1992 (Mycobacterium Infections) : In particular, IL-1 (-alpha and -beta), IL-2, TNF (-alpha and -beta) , and IFN-gamma mRNA were readily detected , whereas IL-3 , IL-4, IL-5, IL-6, IL-7, and granulocyte-macrophage colony stimulating factor mRNA were not detected, or were detectable only at very low levels
Jana et al., Immunology 2009 : Induction of lymphotoxin-alpha by interleukin-12 p40 homodimer, the so-called biologically inactive molecule, but not IL-12 p70
Som et al., Clinical and vaccine immunology : CVI 2012 (Disease Models, Animal...) : Anti-inflammatory SMAMPs prevented the induction of tumor necrosis factor (TNF), interleukin 6 (IL-6), and IL-10 in response to S. aureus or LTA , but no other TLR2 ligands
Palladino et al., Ciba Found Symp 1987 (Neoplasms, Experimental) : Apart from their direct cytotoxic/cytostatic activities against tumours in vitro and in vivo, the in vivo antitumour activities of TNF-alpha or TNF-beta may involve the following biological activities : the induction of interleukin 1 production ; activation of polymorphonuclear neutrophil functions ; modulation of endothelial cell functions ; and augmentation of specific immune functions
Broudy et al., J Immunol 1987 (Inflammation) : We compared the effect of purified recombinant human TNF-alpha and TNF-beta on neutrophil adhesion molecule expression and hematopoietic growth factor production by cultured human umbilical vein endothelial cells
Sugiyama et al., Infect Immun 1996 : In contrast, LPS induced interleukin 1 activity in human gingival epithelial cells in culture, while LTA had little effect
Worm et al., Immunology 1998 (Hypersensitivity) : Stimulation of human B cells with anti-CD40 + interleukin-4 ( IL-4 ) results not only in proliferation and immunoglobulin E ( IgE ) -production, but also increased production of the cytokine lymphotoxin-alpha (LT-alpha) ( formerly also known as tumour necrosis factor-beta (TNF-beta) )