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ICAM1 — ICAM5

Text-mined interactions from Literome

Zhou et al., Mech Ageing Dev 2006 : In this report, ICAM-1 accumulates in late passage endothelial cells when compared to early passage endothelial cells, yet ICAM-1 protein expression is attenuated when senescent cells are challenged by TNF-alpha ( 10ng/ml )
Tiisala et al., Scand J Immunol 1994 : In this study the authors investigated whether a quantitative increase in the surface expression of ICAM-ligands also can lead to enhanced binding to purified ICAM-1
Bryant et al., Infect Immun 1996 : PLC strongly induced expression of ELAM-1, ICAM-1 , and IL-8, while PFO stimulated early ICAM-1 expression but did not promote ELAM-1 expression or IL-8 synthesis
Shen et al., J Virol 1997 : TNF-alpha significantly enhanced WNV induced increases in E-selectin, P-selectin, ICAM-1 , and VCAM-1 expression, while IFN-gamma enhanced WNV induced ICAM-1 expression
Braun et al., Arterioscler Thromb Vasc Biol 1997 (Second Messenger Systems) : In cultured human smooth muscle cells from coronary arteries and saphenous veins, tumor necrosis factor-alpha (TNF-alpha) and interleukin-1 beta (IL-1 beta) induced the expression of intercellular adhesion molecule ( ICAM-1 ) and vascular cell adhesion molecule ( VCAM-1 ), whereas interferon-gamma (INF-gamma) selectively stimulated the expression of ICAM-1
Miyatake et al., Nephron 1998 (Disease Models, Animal) : Although the expression of ICAM-1 can be induced by the stimulation of inflammatory cytokine, ICAM-1 expression can also be induced by such nonimmune mechanisms as shear stress