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CDH1 — CDH4

Text-mined interactions from Literome

Troxell et al., J Cell Sci 2000 : Mutant E-cadherin overexpression caused a dramatic reduction in endogenous cadherin levels
Kintner et al., Cell 1992 : Analysis of the mutant phenotype shows that at least two regions of the N-cadherin cytoplasmic domain can inhibit adhesion and that the mutant cadherin can inhibit catenin binding to E-cadherin
Liu et al., J Cell Biol 2006 : Experiments using microengineered substrates showed that E-cadherin engagement stimulated the peak in proliferation at intermediate seeding densities, and that the proliferation arrest at high densities did not involve E-cadherin , but rather resulted from a crowding dependent decrease in cell spreading against the underlying substrate
Maeda et al., Oncogene 2006 (Skin Neoplasms) : Here, we show that the degradation of E-cadherin in response to expression of R-cadherin is owing to competition for p120(ctn)
Macpherson et al., Oncogene 2007 (Carcinoma, Squamous Cell...) : We show that the collective invasion of A431 cells depends on the presence of cadherin mediated ( P- and E-cadherin ) cell-cell contacts, which are lost in cells where p120 expression is knocked down
Fu et al., Neuro Oncol 2013 : Finally, NPV-LDE-225 suppressed epithelial-mesenchymal transition by upregulating E-cadherin and inhibiting N-cadherin , Snail, Slug, and Zeb1 through modulating the miR-200 family
Nanta et al., Oncogenesis 2013 : NVP-LDE-225 suppressed EMT by upregulating E-cadherin and inhibiting N-cadherin , Snail, Slug and Zeb1 by regulating the miR-200 family
Shimazui et al., Cancer Res 1996 (Urinary Bladder Neoplasms) : In model systems, it has been shown that cadherin function requires not only proper E-cadherin expression but also its linkage to the cytoskeleton through catenins