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CCL26 — TNF

Text-mined interactions from Literome

Hoeck et al., J Immunol 2001 : Although both eotaxin-1 and -3 are regulated by this transcription factor, the response of the eotaxin-3 gene to TNF-alpha stimulation appears to be different
Banwell et al., Cytokine 2002 : Unlike Eotaxin-1, Eotaxin-3 mRNA expression was not induced by either tumour necrosis factor (TNF)-alpha or interleukin (IL)-1 beta alone
Kagami et al., Clin Exp Immunol 2005 (Dermatitis, Atopic) : Furthermore, TNF-alpha generated a synergistic effect on IL-4 enhanced CCL26 production
Rokudai et al., Biol Pharm Bull 2006 (Asthma...) : Differential regulation of eotaxin-1/CCL11 and eotaxin-3/CCL26 production by the TNF-alpha and IL-4 stimulated human lung fibroblast ... We observed that a human lung fibroblast, HFL-1 produces eotaxin-1 and -3 in response to TNF-alpha plus IL-4 stimulation, accompanied with NF-kappaB and STAT6 activation
Igawa et al., Allergy 2006 (Dermatitis...) : Dermal fibroblasts produced eotaxin and eotaxin-3 in response to stimulation by interleukin (IL)-4 and/or tumor necrosis factor-alpha
Stubbs et al., Immunology 2010 : Tumour necrosis factor-alpha (TNF-alpha) and interleukin-1beta (IL-1beta) alone did not induce CCL26 expression, yet these pro-inflammatory cytokines synergized with IL-4 to increase CCL26 protein expression