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CAMK4 — FOS
Text-mined interactions from Literome
Kasahara et al., J Biol Chem 2001
:
CaMKIV was transiently activated in neuronal nuclei after HFS, and the activation returned to the basal level within 30 min. Phosphorylation of CREB, which is a CaMKIV substrate, and expression of
c-Fos protein, which is regulated by CREB,
increased during LTP
Song et al., Cell Signal 2008
(Neuroblastoma) :
Furthermore, we found that OGT is involved in depolarization induced and
CaMKIV dependent activation of
activator protein-1 (AP-1) and subsequent tissue inhibitor of metalloproteinase-1 ( Timp-1 ) gene expression ... Taken together, our findings suggest that CaMKIV activated OGT, and OGT has an essential role on the process of
CaMKIV dependent
AP-1 activation under depolarization in neuronal cells
Guo et al., J Immunol 2008
(Calcium Signaling...) :
Ionomycin also activates
CaMKIV , which, together with p38,
induces AP-1
Ho et al., J Exp Med 1996
:
Here we show that
CaMKIV/Gr induces the transcription factor
activation protein 1 (AP-1) alone or in synergy with T cell mitogens and with the p21ras oncoprotein