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AHSA1 — MAP2K4

Text-mined interactions from Literome

Li et al., Toxicol Appl Pharmacol 1999 : SEK1 phosphorylates and activates both p38 and c-Jun NH ( 2 ) -terminal kinase ( JNK ), whereas MKK3 and MKK6 selectively phosphorylate and activate p38 ... The results presented here demonstrate that endogenous SEK1 does play an important role in the activation of p38 by palytoxin in specific cell types
Xi et al., J Biol Chem 2001 : Here we report that 1 ) p38 mitogen activated protein kinase ( MAPK ) and mitogen activated protein kinase kinase 3 ( MKK3 ) were activated by AICAR in Clone 9 cells, which express only the GLUT1 transporters, and 2 ) activation of p38 was required for AICAR stimulated glucose transport since treatment of the cells with p38 inhibitor SB203580 or overexpression of dominant negative p38 mutant inhibited glucose transport
Taylor et al., Cancer Lett 2008 (Neoplasm Metastasis...) : However, evidence continues to accumulate that JNKK1/MKK4 is also a physiologic activator of p38 under certain conditions, and that activation of p38 arrests cell cycle progression
Huang et al., Appl Microbiol Biotechnol 2012 : Besides, EPMC also significantly suppressed the phosphorylation of p38 mitogen activated protein kinase ( p38 ), c-jun N-terminal kinase, mitogen activated protein kinase kinase 4, and expression of NF-?B inducing kinase, while IPMC seemed to show no regular effect on p38