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JUN — VAV1
Protein-Protein interactions - manually collected from original source literature:
Studies that report less than 10 interactions are marked with *
Text-mined interactions from Literome
Kaminuma et al., Mol Cell Biol 2001
:
On the other hand,
Vav induced the activation of Rac1 or Cdc42 and
c-Jun N-terminal kinase (JNK) , enhanced the transcriptional and DNA binding activities of AP-1, and induced increased phosphorylation of c-Jun ... On the other hand,
Vav induced the activation of Rac1 or Cdc42 and c-Jun N-terminal kinase (JNK),
enhanced the transcriptional and DNA binding activities of
AP-1 , and induced increased phosphorylation of c-Jun
Kaminuma et al., FEBS Lett 2002
:
Vav enhanced TCR induced activation of
c-Jun N-terminal kinase (JNK) and its upstream regulator, Rho family GTPases
Feito et al., Eur J Immunol 2003
(MAP Kinase Signaling System) :
Anti-H4/ICOS antibodies strongly enhanced CD3 mediated tyrosine phosphorylation of ZAP-70, zeta, or
Vav , as well as extracellular signal regulated kinase ( ERK ),
Jun N-terminal kinase (JNK) and p38 MAP kinase
activation in these cells
Bustelo et al., Oncogene 1997
:
Cbl-b, a member of the Sli-1/c-Cbl protein family, inhibits
Vav mediated
c-Jun N-terminal kinase activation
Miranti et al., Curr Biol 1998
:
Additionally, Syk and
Vav1 cooperatively
induced activation of
Jun N-terminal kinase (JNK) , extracellular-signal regulated kinase 2 ( ERK2 ) and the kinase Akt, and phosphorylation of the oncoprotein Cbl in fibrinogen-adherent cells