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CASP16 — ICAM1
Text-mined interactions from Literome
Choi et al., Neurosci Lett 2003
(Astrocytoma...) :
Studies using Boc-D-Fmk, a pharmacological inhibitor, show that
caspase activation is
required for Fas mediated
ICAM-1 induction
Takahashi et al., J Immunother 2005
:
Although caspase-1 inhibitor completely abolished the production of IL-18, anti-IL-18 mAb and
caspase-1 inhibitor partially
inhibited the increase in
ICAM-1 and CD40 expression induced by doxazosin, prazosin, and terazosin
Fawcett et al., Int J Oncol 2005
(Breast Neoplasms) :
We conclude that the paclitaxel induced reduction in
ICAM-1 expression by MDA-MB-435 breast carcinoma cells is both ROS- and
caspase dependent , whereas paclitaxel induced cytotoxicity is ROS dependent and does not involve caspases
Tang et al., Zhong Xi Yi Jie He Xue Bao 2007
(Infarction, Middle Cerebral Artery...) :
The mRNA expression level of
caspase-3 in brain tissue in the PNS group was significantly lower than that in the untreated group ( P < 0.05 ), but PNS
had no effect on the mRNA expression levels of
ICAM-1 , caspase-1 and caspase-8 in brain tissue
Gustavsson et al., Retina 2008
(Diabetes Mellitus, Experimental...) :
Ischemia induced increased expressions of TNF-alpha ( P < or= 0.012 ), IL-1beta ( P < or= 0.017 ),
ICAM-1 ( P < or= 0.025 ), and IL-6 ( P = 0.012 ), and diabetes
induced increased expression of
caspase-1 ( P < or= 0.046 ), VCAM-1 ( P < or= 0.027 ), ICAM-1 ( P < or= 0.016 ), IL-1beta ( P = 0.016 ), and IL-6 ( P = 0.041 )