◀ Back to SETD2

SETD2 — TCF19

Text-mined interactions from Literome

Vincent et al., Circulation 2000 (Ischemia) : After transfection into HeLa, C6, and Hep3B cells, this chimeric transcription factor was shown to activate expression of the endogenous VEGF gene, as well as several other HIF-1 target genes in vitro
Ruscher et al., J Neurosci 2002 (Hypoxia-Ischemia, Brain) : In astrocytes the OGD enhanced the nuclear translocation of hypoxia-inducible factor 1 (HIF-1) , the major transcription factor regulating EPO expression
Kirito et al., Cell cycle (Georgetown, Tex.) 2005 : Like several other effects of the cytokine, the effect of TPO on VEGF expression is mediated by stabilization and activation of the primary transcription factor responsible for VEGF expression, the oxygen tension responsive hypoxia inducible factor-1 (HIF-1)
Xie et al., Cell Signal 2008 : Here we show that SETD2 could interact with p53 and selectively regulate the transcription factor activity of p53
Díez et al., Br J Pharmacol 2010 (Inflammation) : Nitric oxide ( NO ) modulates expression of hypoxia inducible factor-1 (HIF-1) , a transcription factor regulating function of myeloid cells
Haddad et al., Curr Mol Pharmacol 2013 (Anoxia) : The NMDAR connection with hypoxia-inducible factor-1a (HIF-1a) , a transcription factor considered master regulator of oxygen sensing mechanisms, is not well established in the CNS