Zhang et al., J Biol Chem 2005
(Breast Neoplasms...) :
Overexpression of NFAT3 enhanced both ERalpha and ERbeta transcriptional activities in a ligand independent manner and up-regulated downstream estrogen-responsive genes including pS2 and cathepsin D
Zhang et al., Biochem Biophys Res Commun 2007
(Wilms Tumor) :
Replacement of Ser168 and Ser170, the amino acid residues on which NFAT3 can be phosphorylated, with Ala did not change the ability of NFAT3 to inhibit the transcriptional activity of ERalpha and ERbeta