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DNAJA3 — EGFR

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

• IRef Biogrid Interaction: EGFR — DNAJA3 (physical association, affinity chromatography technology) Chen et al., Cancer Res 2013*
• IRef Biogrid Interaction: EGFR — DNAJA3 (physical association, affinity chromatography technology) Foerster et al., Proteomics 2013
• IRef Intact Interaction: Complex of 70 proteins (association, anti bait coimmunoprecipitation) Thelemann et al., Mol Cell Proteomics 2005

Text-mined interactions from Literome

Chen et al., J Pathol 2009 (Carcinoma, Squamous Cell...) : Thirdly, we showed that overexpression of Tid1 attenuated EGFR activity and blocked the activation of AKT in HNSCC cells, which are known to be involved in the regulation of survival in HNSCC cells
Chen et al., Cancer Res 2013 (Adenocarcinoma...) : Tid1-L overexpression in lung cancer cells attenuated EGFR signaling and inhibited cell proliferation, colony formation, and tumor growth in subcutaneous and orthotropic xenograft models ... Conversely, depletion of Tid1 restored EGFR signaling and increased cell proliferation and colony formation ... Tid1-L inhibited EGFR signaling even more than the HSP90 inhibitor 17-allylamino-demethoxy geldanamycin