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EGFR — POLDIP2

Text-mined interactions from Literome

Kanda et al., Br J Pharmacol 2001 : In the present study, we examine the involvement of epidermal growth factor (EGF) receptor in thrombin induced p38 MAPK activation
Funakoshi et al., J Biol Chem 2002 : Inhibition of epidermal growth factor-receptor by AG1478 suppressed the AngII induced CREB phosphorylation as well as activation of ERK and p38MAPK
Seo et al., J Biol Chem 2004 : UV-induced p38 activation was inhibited by N17 Ras and a farnesyltransferase inhibitor, manumycin A. Gbetagamma also increased the UV-induced phosphorylation of the epidermal growth factor receptor (EGFR), and the UV-induced p38 activation was blocked by an EGFR kinase inhibitor, AG1478
Cui et al., Free Radic Biol Med 2006 : We observed that inhibition of EGFR kinase activity with AG1478 significantly attenuated H2O2 induced p38 ( SAPK ) and ERK1/2 activation, but did not inhibit Akt1 activation
Shin et al., Biochim Biophys Acta 2009 : Cross interference with TNF-alpha induced TAK1 activation via EGFR mediated p38 phosphorylation of TAK1 binding protein 1
Zhou et al., Zhonghua Yi Xue Za Zhi 2012 (MAP Kinase Signaling System) : The cells were pre treated with antibodies of ErbB1-4 , inhibitors of p38 mitogen activated protein kinase ( MAPK ), ERK1/2, mitogen- and stress activated protein kinase ( MSK ) 1 and antibody of cAMP-response element binding protein ( CREB )