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PI3 — VCAM1

Text-mined interactions from Literome

Morel et al., J Biol Chem 2001 (Arthritis, Rheumatoid) : IL-18 requires NF kappa B as well as PI 3-kinase to induce VCAM-1 on RA synovial fibroblasts, suggesting that there may be two distinct pathways in IL-18 induced adhesion molecule expression
Amin et al., Blood 2006 : Antisense oligodeoxynucleotides ( ODNs ) and inhibitors of Src, PI3K , p38, and NFkappaB significantly reduced rhMIF induced MN VCAM-1 and ICAM-1 expression ( P < .05 )
Hosokawa et al., Clin Exp Immunol 2006 (Periodontitis) : Phosphatidylinositol 3-kinase (PI3K) and nuclear factor-kappaB (NF-kappaB) inhibitor inhibit both ICAM-1 and VCAM-1 expression induced by TWEAK
Tung et al., Cell Signal 2007 : In this study, we identified a requirement for PDGFR, PI3-K/Akt , p38 MAPK, JNK, and NF-kappaB in the regulation of VCAM-1 expression by rat vascular smooth muscle cells ( VSMCs ) in response to viral infection
Binion et al., Am J Physiol Gastrointest Liver Physiol 2009 : Tumor necrosis factor (TNF)-alpha/lipopolysaccharide (LPS) induced VCAM-1 expression in HIMEC was suppressed by Akt small interfering RNA, curcumin, and inhibitors of NF-kappaB ( SN-50 ), p38 MAPK ( SB-203580 ) and PI 3-kinase/Akt ( LY-294002 )