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NOS2 — NPS
Text-mined interactions from Literome
Hu et al., Biomaterials 2010
(Body Weight) :
Moreover, TiO ( 2 )
NPs significantly
inhibited the activities of Na ( + ) /K ( + ) -ATPase, Ca ( 2+ ) -ATPase, Ca ( 2+ ) /Mg ( 2+ ) -ATPase, acetylcholine esterase, and
nitric oxide synthase ; the function of the central cholinergic system was also noticeably disturbed and the contents of some monoamines neurotransmitters such as norepinephrine, dopamine and its metabolite 3, 4-dihydroxyphenylacetic acid, 5-hydroxytryptamine and its metabolite 5-hydroxyindoleacetic acid were significantly decreased, while the contents of acetylcholine, glutamate, and nitric oxide were significantly increased
Li et al., Biol Trace Elem Res 2012
(Disease Resistance) :
In addition, the TiO ( 2 )
NPs significantly
promoted the expression of resistance related genes, including those encoding superoxide dismutase, catalase, glutathione peroxidase, acetylcholine esterase, carboxylesterase, heat shock protein 21, glutathione S transferase o1, P53, and transferring and of genes encoding cytochrome p302 and
nitric oxide synthase
Liu et al., Biomaterials 2013
(Brain Ischemia) :
Furthermore,
CBSA-PEG-TIIA-NPs significantly decreased the mRNA expressions of iNOS and p38MAPK, upregulated PPAR? expression, and
inhibited the protein levels of
iNOS , GFAP and p38MAPK phosphorylation