Ribaux et al., Endocrinology 2007
:
Induction of CXCL1 by extracellular matrix and autocrine enhancement by interleukin-1 in rat pancreatic beta-cells
Heinhuis et al., Ann Rheum Dis 2011
(Arthritis, Rheumatoid) :
TNFa stimulation of FLS after silencing IL-32? resulted in diminished IL-6 and CXCL8 production, whereas IL-32? overexpression resulted in enhanced IL-6 and CXCL8 levels ... Remarkably, the mechanism through which IL-32? overexpression induced TNFa, IL-1ß and CXCL8 was by counteracting messenger RNA decay