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FOS — PDCD11
Text-mined interactions from Literome
Lehmann et al., Cell Death Differ 2002
:
This study demonstrates a
role for
AP-1 in the stage-specific steroid triggered
programmed cell death of larval tissues during Drosophila metamorphosis
ZahradnĂcková et al., Cell Mol Life Sci 2003
(Cell Transformation, Neoplastic...) :
c-Fos but not v-Fos protein
induces programmed cell death of v-myb transformed monoblasts ... We show that while the v-Fos protein prolongs the G0G1 phase of the BM2 cell cycle,
c-Fos leaves the cell cycle unaffected and, rather,
induces programmed cell death
Zhu et al., Int J Biochem Cell Biol 2012
(Bone Resorption) :
The RANKL stimulated mRNA expression of osteoclast related genes and transcription factors were also diminished by ISL. Mechanistically, ISL blocked the RANKL triggered RANK-TRAF6 association, phosphorylation of mitogen activated protein kinases ( MAPKs ), inhibitor of ?Ba ( I?Ba ) phosphorylation and degradation,
nuclear factor-?B ( NF-?B ) p65 nuclear translocation, as well as
activator protein (AP)-1 activation
Marti et al., Oncogene 1994
:
A similar induction of AP-1 ( cFos/JunD ) was also observed in the involuting rat ventral prostate pointing to a possible
role for
AP-1 in
programmed cell death
Feng et al., Oncogene 1998
(Atrophy) :
Our data strongly suggest that
c-Fos is
required for
programmed cell death of prostate epithelial cells