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FOS — IGF1

Text-mined interactions from Literome

Xu et al., J Neuropathol Exp Neurol 2001 (Diabetes Mellitus, Type 1...) : The delayed IGF-1 expression may affect C-FOS induction and may be responsible for the delay in the NGF response in diabetic rats
Kim et al., Biochem Biophys Res Commun 2004 (MAP Kinase Signaling System) : Furthermore, IGF-II remarkably increased the DNA binding activities of NF-kappaB and AP-1 , and the IL-8 promoter activity
Freiss et al., J Steroid Biochem Mol Biol 2005 (Breast Neoplasms) : We have tested the effects of two Eli-Lilly compounds, LY 117, 018 and raloxifene, on E2-regulated and IGF-I induced proliferation or AP-1 activity in human breast cancer cells ... Moreover, raloxifene was the most efficient molecule to prevent IGF-I induced AP-1 activity, with a significant effect observed with a concentration as low as 5 x 10 ( -11 ) M in the presence of IGF-I alone
Jia et al., J Cell Mol Med 2011 : These data demonstrate a cross-talk between IGF-1R and AT-1R in AT-II and IGF-1 induced Cx43 expression in SV SMCs involving Erk 1/2 and downstream activation of the AP-1 transcription factor
Yang et al., Biochem J 2011 (Colonic Neoplasms) : IGF-1 induced MAT2A promoter activity and increased nuclear protein binding to USF ( upstream stimulatory factor ) /c-Myb, YY1 ( Yin and Yang 1 ), E2F, AP-1 ( activator protein 1 ) and NF-?B ( nuclear factor ?B ) consensus elements
Puzik et al., Cytokine 2012 (Infection) : Furthermore the effect of IGF-I on cytokine expression, apoptosis and the DNA binding activity of AP-1 and NF?B was evaluated
Torres-Aleman et al., Neuroscience 1998 : In addition, insulin-like growth factor-I increased intracellular Ca2+ levels in Purkinje cells and c-Fos in dividing glioblasts