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AVP — IRF6

Text-mined interactions from Literome

Umino et al., Am J Physiol 1999 : AVP inhibits LPS- and IL-1beta stimulated NO and cGMP via V1 receptor in cultured rat mesangial cells ... AVP inhibited lipopolysaccharide (LPS)- and interleukin-1beta (IL-1beta) induced nitrite production in a dose- and time dependent manner, with concomitant changes in cGMP content, iNOS mRNA, and iNOS protein ... In conclusion, AVP inhibits LPS- and IL-1beta induced NO production through a V1 receptor
Mancuso et al., J Neuroimmunol 1999 : SnPP9 given i.p. failed to modify either basal or LPS stimulated levels of AVP and Cort
Matsunaga et al., Brain Res 2000 : The present data suggest that LPS induced peripheral release of AVP and OXT is due to the activation of the magnocellular neurons in the SON, magPVN, NC, and rSON, and the central release of those hormones is in part derived from the activation of parvocellular neurons in the PVN
Wei et al., J Neuroimmunol 2002 (Inflammation) : However, LPS induced AVP release and ACh evoked CRH secretion in LEW/N were comparable with those of F344/N
Shimizu et al., J Appl Physiol 2009 (Disease Models, Animal...) : LPS induced AVP secretion was significantly attenuated by pretreatment with icv losartan ( 2.3 +/- 0.5 to 3.7 +/- 0.5 pg/ml ) but was not attenuated after peripheral captopril treatment ( 2.2 +/- 0.6 to 17.6 +/- 4.2 pg/ml )
Evuarherhe et al., J Physiol 2009 (Body Weight) : Testosterone significantly suppressed the corticotrophin releasing hormone and arginine vasopressin mRNA as well as the corticosterone response to LPS in castrated animals that had had their testes intact over puberty
Leggett et al., J Neuroendocrinol 2009 (Disease Models, Animal...) : Parvocellular paraventricular nucleus ( PVN ) corticotrophin releasing factor (CRF) and corticotrophic pro-opiomelanocortin (POMC), but not parvocellular PVN arginine vasopressin (AVP) , mRNA expression was significantly increased by LPS compared to non-LPS control
Parkes et al., Life Sci 1995 (Hypertension) : The present study investigated the effects of both intravenous ( IV ) and intracerebroventricular ( ICV ) injection of lipopolysaccharide (LPS) from E. coli on body temperature ( Tb ), mean arterial pressure ( MAP ), heart rate ( HR ), cardiac output ( CO ), calculated total peripheral resistance ( CTPR ), stroke volume ( SV ) and plasma levels of adrenocorticotropin ( ACTH ) and arginine vasopressin (AVP) in conscious, chronically instrumented sheep
Kjaer et al., Neuroendocrinology 1995 (Hypoglycemia) : Restraint stress and HA but not LPS or insulin stress induced an increase in peripheral OT levels, whereas only LPS stress and HA caused an increase in circulating AVP levels