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RNF19A — SLPI

Text-mined interactions from Literome

Suzuki et al., Bone 2002 : Distinct MAPK pathways seem to independently modulate osteoblastic cell proliferation and differentiation, with Erk playing an essential role in cell replication, whereas p38 is involved in the regulation of ALP expression during osteoblastic cell differentiation
Kakita et al., Prostaglandins Leukot Essent Fatty Acids 2004 : Recently, we have shown that p38 mitogen activated protein ( MAP ) kinase pathway regulates ALP activity in response to activation of Gi protein coupled receptors in mouse osteoblast-like MC3T3-E1 cells ( Suzuki et al., Endocrinology 140 ( 1999 ) 3177 )
Rey et al., Bone 2007 (MAP Kinase Signaling System) : Interestingly, PTH induced ALP stimulation was dose-dependently inhibited by a specific p38 inhibitor with no change in the generation of cAMP and the production of osteocalcin
Qin et al., Int Endod J 2012 : BMP-2 dose- and time-dependently upregulated phosphorylation of p38a of HDPCs. Compared with BMP-2-treatment group, gene knock-down of p38a MAPK significantly inhibited ALP activity and the formation of mineralized nodules in HDPCs