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RNF19A — SOD1
Protein-Protein interactions - manually collected from original source literature:
Studies that report less than 10 interactions are marked with *
-
IRef Biogrid Interaction:
RNF19A
—
SOD1
(physical association, affinity chromatography technology)
Niwa et al., J Biol Chem 2007*
-
IRef Biogrid Interaction:
RNF19A
—
SOD1
(physical association, affinity chromatography technology)
Niwa et al., J Biol Chem 2002*
-
IRef Biogrid Interaction:
RNF19A
—
SOD1
(direct interaction, enzymatic study)
Niwa et al., J Biol Chem 2002*
-
IRef Biogrid Interaction:
RNF19A
—
SOD1
(physical association, affinity chromatography technology)
Ishigaki et al., Neurobiol Dis 2007*
-
IRef Hprd Interaction:
SOD1
—
RNF19A
(in vivo)
Niwa et al., J Biol Chem 2002*
-
IRef Hprd Interaction:
SOD1
—
RNF19A
(in vitro)
Niwa et al., J Biol Chem 2002*
Text-mined interactions from Literome
Pavlovic et al., Eur Cytokine Netw 2000
(Diabetes Mellitus, Type 1) :
When added together,
p38i and MEKi decreased IL-1 beta induced nitrite production over 24 hours by 60 %, but did not
affect IL-1 beta induced manganese
superoxide dismutase ( MnSOD ) mRNA expression
Niwa et al., J Biol Chem 2002
:
Dorfin physically bound and ubiquitylated various SOD1 mutants derived from familial ALS patients and enhanced their degradation, but it
had no effect on the stability of the wild-type
SOD1
Gaitanaki et al., J Exp Biol 2008
:
Hyperthermia induced
p38-MAPK activation was found to be dependent on the Na+/H+ exchanger 1 (NHE1) and was also
suppressed by catalase (Cat) and
superoxide dismutase ( SOD ) , implicating the generation of reactive oxygen species ( ROS )