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APP — GSK3B

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

• IRef Biogrid Interaction: GSK3B — APP (physical association, affinity chromatography technology) Zhou et al., Biochim Biophys Acta 2012*
• IRef Biogrid Interaction: GSK3B — APP (direct interaction, pull down) Oláh et al., J Biol Chem 2011

Text-mined interactions from Literome

Kim et al., FASEB J 2003 : Moreover, expression of APP770 and Swedish mutant form of APP increased the levels of C-terminal fragments of APP ( APP-CTs ) in neuronal cells and also induced the up-regulation of glycogen synthase kinase-3beta at both the mRNA and the protein levels
Lin et al., Neurobiol Aging 2009 : Furthermore, EGCG also decreased nuclear translocation of c-Abl and blocked APP-C99 dependent GSK3 beta activation , and these inhibitory effects occurred through the interruption of c-Abl/Fe65 interaction
Lau et al., J Biol Chem 2008 : We also demonstrate that Dexras1 inhibits the FE65-APP mediated transcription of glycogen synthase kinase 3beta ( GSK3 beta )
Takashima et al., Neurosci Lett 1995 : These results provide evidence that APP accumulation after A beta treatment is regulated by TPK I/GSK-3 beta