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FES — STAT3

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

• IRef Biogrid Interaction: STAT3 — FES (physical association, affinity chromatography technology) Park et al., Cancer Lett 1998*
• IRef Hprd Interaction: STAT3 — FES (in vitro) Park et al., Cancer Lett 1998*
• IRef Hprd Interaction: STAT3 — FES (in vivo) Park et al., Cancer Lett 1998*
• IRef Ophid Interaction: STAT3 — FES (aggregation, interologs mapping) Brown et al., Bioinformatics 2005

Text-mined interactions from Literome

Kim et al., J Biol Chem 2003 : Further analysis showed that Fes ( act ) caused activation of CCAAT/enhancer binding protein-alpha and STAT3 , two transcription factors that are involved in granulocyte differentiation
Nelson et al., J Biol Chem 1998 : In this report, we provide direct evidence that c-Fes , the normal human homolog of v-Fps, potently activates STAT3 ... To determine whether Fes induced STAT3 activation is dependent upon endogenous mammalian kinases, co-expression studies were also performed in Sf-9 insect cells ... Fes also induced a dramatic increase in STAT3 DNA binding activity in this system, whereas no activation of STAT5 was observed ... Fes induced strong tyrosine phosphorylation of STAT3 in both expression systems, consistent with the gel-shift results