◀ Back to EGF

EGF — NPY6R

Text-mined interactions from Literome

Samet et al., Toxicol Appl Pharmacol 2003 : Exposure to Zn ( 2+ ), but not EGF , induced phosphorylation of the activating site of c-Src ( tyrosine 416 ), and Zn ( 2+ ) -induced phosphorylation of EGFR at tyrosines 845 and 1068 was blocked by the c-Src kinase activity inhibitor PP2
Xu et al., Invest Ophthalmol Vis Sci 2007 : The release of HB-EGF assessed by AP activity increased significantly in response to wounding, LPA, or both, and the release of HB-EGF-AP induced by LPA was inhibited by PP2 and GM6001
Lin et al., Am J Physiol Cell Physiol 2008 : The Src inhibitor PP2 blocked pilocarpine induced ERK activation and EGF receptor phosphorylation, without affecting ERK activation by carbachol
Suh et al., J Cell Physiol 2009 : In addition, IL-6 increased EGFR/src/FAK, PI3K/Akt phosphorylation and 2-DG uptake as well as GLUT-2 protein expression, which were blocked by AG 1478 ( EGF receptor inhibitor ), PP2 ( src family of tyrosine kinase inhibitor ), PI3K-specific siRNA, and a Akt inhibitor
Dong et al., Biochim Biophys Acta 2011 : PP2 , a c-Src specific inhibitor, suppressed both TCDD- and epidermal growth factor- (EGF) induced c-Src activation