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IL1A — IL3

Text-mined interactions from Literome

De Caterina et al., Am J Clin Nutr 2000 (Arteriosclerosis) : Consumption of the n-3 fatty acid docosahexaenoic acid ( DHA ; 22 : 6n-3 ) reduced endothelial expression of vascular cell adhesion molecule 1 ( VCAM-1 ), E-selectin, intercellular adhesion molecule 1 ( ICAM-1 ), interleukin 6 (IL-6), and IL-8 in response to IL-1 , IL-4, tumor necrosis factor, or bacterial endotoxin, with a half-maximal inhibitory concentration ( IC ( 50 ) ) of 1-25 micromol, ie, in the range of nutritionally achievable plasma concentrations
Zeyse et al., Biol Reprod 2000 : Induction of interleukin-1alpha production in murine Sertoli cells by interleukin-1
Hültner et al., J Immunol 2000 : Here we report that in primary mouse bone marrow derived mast cells activated by ionomycin or IgE/Ag, the proinflammatory mediator IL-1 ( alpha or beta ) up-regulated production of IL-3 , IL-5, IL-6, and IL-9 as well as TNF, i.e., cytokines implicated in many inflammatory processes including those associated with allergies and helminthic infections
Hestdal et al., Blood 1992 : Pretreatment of mice with anti-type I IL-1 receptor antibody blocked the IL-1 alpha induced upregulation of GM-CSF and IL-3 receptor expression on BMCs. Taken together, as one possible mechanism, IL-1 alpha in vivo may stimulate the expression of functional GM-CSF and IL-3 receptors on BMCs indirectly, and, in concert with the induction of circulating CSF levels, may account for the ability of IL-1 alpha to stimulate hematopoiesis in vivo
Walker et al., J Immunol 1992 (Mycobacterium Infections) : The effect of in vitro exposure of these cells to the immunizing Ag was also investigated ; again, IL-1 , IL-2, TNF, and IFN-gamma mRNA were abundant, but in addition, IL-3 , IL-6, and granulocyte-macrophage colony stimulating factor mRNA were greatly increased , suggesting an important role in the recall response
Beyaert et al., Cytokine 1991 : This study shows the in vitro effect of LiCl on the TNF induced or interleukin 1 (IL-1) induced expression of IL-6, granulocyte-macrophage colony stimulating factor ( GM-CSF ), IL-3 , IL-2, and the IL-2 receptor-alpha ( IL-2R alpha ) ... However, LiCl potentiated the IL-1 induced synthesis of IL-6, GM-CSF, IL-3 , and IL-2 in PC60 murine T-cell hybridoma cells
Van Damme et al., J Invest Dermatol 1990 : This study focuses on interferon-beta (IFN-beta), interleukin-6 (IL-6), and interleukin-8 (IL-8) production by fibroblasts and epithelial cells in response to interleukin-1 (IL-1) and tumor necrosis factor-alpha (TNF-alpha)
Cohen et al., Immunol Lett 1991 : We have studied the production of interleukin-1 (IL-1) , interleukin-6 (IL-6) and tumor necrosis factor (TNF) by mouse peritoneal macrophages triggered by lipopolysaccharide (LPS) in the presence or absence of IL-3 ... Interleukin-3 at the concentration used ( i.e., 100 U/ml ) did not induce the production of any cytokines, whereas it enhanced significantly the secretion of IL-1 , IL-6 and TNF by LPS stimulated macrophages
Teshima et al., Br J Haematol 1991 (Leukemia, Myelogenous, Chronic, BCR-ABL Positive) : These results suggest that basophil progenitors expressing CD4, CD7 and HLA-DR may be involved in the development of basophilic crisis of CML and that both IL-1 and GM-CSF may act on basophil progenitors as well as IL-3 or IL-4
Elias et al., Chest 1990 (Pneumonia...) : To understand the processes regulating inflammation and fibrosis in the human lung, we characterized the effects of recombinant interleukin-1 , tumor necrosis factor, and gamma interferon on fibroblast proliferation, collagen production, interleukin-1-alpha production, interleukin-1-beta production, and interleukin-6 production
Hart et al., Immunology 1990 : IL-3 alone stimulated monocyte u-PA activity, but not TNF-alpha or IL-1 activity ... However, IL-3 , together with interferon-gamma (IFN-gamma), stimulated the TNF-alpha, but not IL-1 , activities of monocytes from several donors
Cavaillon et al., Cell Immunol 1990 : Kinetic studies of IL-1 induced IL-3 production indicated that 4-6 days of culture were required for optimal production, whereas 1-2 days were sufficient in cultures stimulated with concanavalin A. Recombinant IL-6 failed to induce significant amounts of IL-3, and TNF alpha induced only weak IL-3 production ... Removal of macrophages decreased the production of IL-3 induced by LPS and GMF-CSF though did not affect the IL-3 production induced by IL-1
Luger et al., Immunobiology 1986 : Anti-IL 1 IgG blocked the IL 1-mediated thymocyte and fibroblast proliferation and also inhibited the biological activity of epidermal cell derived thymocyte activating factor ( ETAF ), but did not affect interleukin 2 (IL 2) and interleukin 3 (IL 3) activity
Weiss et al., Eur J Immunol 1989 : IL 2 and IL 3 enhanced IL 1 production by LPS stimulated cells whereas granulocyte-monocyte colony stimulating factor had no effect
Seckinger et al., J Immunol 1987 (Fever) : A urine inhibitor of interleukin 1 activity affects both interleukin 1 alpha and 1 beta but not tumor necrosis factor alpha
Schwarz et al., J Invest Dermatol 1988 : Serum-contra IL 1 did not suppress interleukin 2 or interleukin 3 and did not inhibit spontaneous cell proliferation
Yang et al., J Cell Physiol 1988 : These cells constitutively elaborate hematopoietic growth factor activity into the medium and the level of production of this activity dramatically increases following stimulation of the cells with IL-1
Schwarz et al., J Immunol 1987 : EC-contra-IL 1 also blocked IL 1-induced fibroblast proliferation but did not suppress IL 2 or IL 3 activity
Rameshwar et al., J Immunol 1994 : The induction of IL-3 and GM-CSF is partially mediated by IL-1 and IL-6, which are also produced by bone marrow mononuclear cells
Bruserud et al., Leuk Res 1995 (Leukemia, Myeloid, Acute) : IL-3 and granulocyte/macrophage colony stimulating factor increased secretion of IL-1 alpha, IL-1 beta and TNF-alpha for a majority of AML patients, whereas IL-4 decreased cytokine secretion
Snapper et al., J Immunol 1995 : Although both alpha delta-dex and IL-1 + IL-2 are required for optimal IL-3- and GM-CSF mediated IgM secretion, both IL-3 and GM-CSF stimulate a modest IgM secretory response by cells activated with alpha delta-dex alone
Grosset et al., Blood 1995 : In vitro biosynthesis of leukemia inhibitory factor/human interleukin for DA cells by human endothelial cells : differential regulation by interleukin-1 alpha and glucocorticoids
Hestdal et al., Blood 1994 : Therefore, the synergistic effects of IL-1 alpha on IL-3- , CSF-1-, and granulocyte macrophage (GM)-CSF induced progenitor growth, both in CFU-c and single-cell assays, were determined in the presence of monoclonal antibodies ( MoAbs ) 35F5 and 4E2 that block the binding of IL-1 alpha to type I and type II IL-1R, respectively
Boxman et al., J Invest Dermatol 1993 : Low interleukin-1 activity was most probably not due to inhibition of interleukin-1 alpha production in squamous carcinoma cells-4/3T3 co-cultures because interleukin-1 alpha messenger RNA expression in squamous carcinoma cells-4 cells remained unchanged in the presence of 3T3 cells
Dinarello et al., Nutrition 1995 (Disease) : IL-4, IL-10, and IL-3 suppress gene expression and synthesis of IL-1 and TNF