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Pathways - manually collected, often from reviews:
OpenBEL Selventa BEL large corpus:
PAK1
→
PRKCB
(increases, PRKCB Activity)
Menard et al., Cell Signal 2003* Evidence: Studies using inhibitors of the EGF receptor tyrosine kinase, phosphatidylinositol 3-kinase (PI3-kinase) and protein kinase C (PKC) revealed that all of the cell surface agonists could activate PAK1 through pathways independent of PKC, that EGF stimulated a PI3-kinase dependent pathway to stimulate PAK1, and that muscarinic receptor stimulation of PAK1 was predominantly mediated through this EGF-R-dependent mechanism