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ELAVL1 — MYLIP

Text-mined interactions from Literome

Abdelmohsen et al., Proc Natl Acad Sci U S A 2008 : Importantly, the growth promoting effects of ( AS ) miR-519 required the presence of HuR , because downregulation of HuR by RNAi dramatically suppressed its proliferative action
Guo et al., RNA Biol 2009 (Breast Neoplasms) : Real time PCR and gene reporter assays indicated that HuR was translationally repressed by miR-125a
Glorian et al., Cell Death Differ 2011 : However, we show that miR-19 mediated regulation of antiapoptotic RhoB expression requires the binding of human antigen R (HuR) , an AU-rich element binding protein, to the 3'-untranslated region of the rhoB mRNA
Young et al., Mol Cancer Res 2012 (Carcinoma...) : Furthermore, this interaction between HuR and miR-16 promoted the downregulation of miR-16
Leucci et al., Oncogene 2012 (Hodgkin Disease) : Finally, inhibition of miR-9 by a systemically delivered antimiR-9 in a xenograft model of HL increases the protein levels of HuR and DICER1 and results in decreased tumour outgrowth, confirming that miR-9 actively participates in HL pathogenesis and points to miR-9 as a potential therapeutic target
Prislei et al., BMC cancer 2013 (Adenocarcinoma...) : Through the analysis of 3'UTR associated complexes, we found that the miR-200c can increase the association of the RNA binding protein HuR with TUBB3 mRNA, whereas HuR binding enhanced TUBB3 mRNA translation
Balkhi et al., Science signaling 2013 : By interacting directly with the RNA binding protein HuR ( human antigen R ; also known as ELAVL1 ), miR-29 prevented HuR from binding to the A20 3'UTR and recruiting the RNA degradation complex RISC ( RNA induced silencing complex ), suggesting that miR-29 can act as a decoy for HuR, thus protecting A20 transcripts