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CSF2 — GPI

Text-mined interactions from Literome

Stanford et al., Br J Pharmacol 2000 : These observations suggest that PGI ( 2 ), released following the induction of COX-2, differentially regulates the release of GM-CSF ( suppresses ) and G-CSF ( potentiates ) from human vascular cells
Lenda et al., J Immunol 2003 (Inflammation...) : We suggest that during renal inflammation, CSF-1 mediates M phi recruitment, proliferation, activation, and, in turn, TEC apoptosis
Takeda et al., Exp Cell Res 2003 : Granulocyte colony stimulating factor , which augments neutrophil differentiation of HL-60 cells, up-regulated GPI-80 expression in the presence of DMSO
Rajotte et al., J Biol Chem 1992 (Leukemia, Megakaryoblastic, Acute) : Preincubation of the cells with staurosporine, a PKC inhibitor, also significantly reduces the effect of GM-CSF or IL-3 on pHi
Walker et al., Immunobiology 1992 (Fibrosarcoma) : In the absence of M phi , GM-CSF increased T cell proliferation in response to alloantigen ... The data collectively suggest that TBH M phi respond differently to GM-CSF, and that tumor induced changes in GM-CSF responsiveness affect M phi accessory ability
Bouyer et al., Endocrinology 2007 : The present study shows that, in the absence of CO ( 2 ) /HCO ( 3 ) ( - ), CSF-1 causes little change in osteoclast pH(i)
Kato et al., Cell Immunol 1990 : We investigated the augmentative effect of GM-CSF on M phi A-cell activity in concanavalin A-stimulated T-cell proliferation ... We observed that GM-CSF induced M phi spreading and firm attachment accompanied with enlargement of the cytoplasm, but could not induce de novo expression of Ia antigen ... These results indicate that GM-CSF augments M phi A-cell activity through the enhancement of the capacity of M phi to produce IL-1 and IL-6
Hughes et al., Infect Immun 1987 : Thus, rGM-CSF does not induce adequate M phi activation to kill T. gondii and is not a major microbistatic component of ConAS
Fischer et al., J Immunol 1988 : The activation of BMM phi by GM-CSF was reversible and could be repeated
Gessl et al., Immunobiology 1995 : Furthermore, GM-CSF was able to trigger expression of class II molecules on rat peritoneal macrophages ( M phi ) and BMDM phi resulted from cultures of BMC with mouse M phi-CSF (M-CSF), and the RT1.B and RT1.D inducing effect of GM-CSF was opposed by M-CSF, and by anti-GM-CSF antibodies
Chen et al., J Immunol 1994 (Cryptococcosis) : GM-CSF induced AM phi proliferation, but anticryptococcal activity was not due totally to increases in AM phi numbers, indicating AM phi activation by GM-CSF
Sasaki et al., Blood 1997 (Hematologic Neoplasms...) : Phorbol 12-myristate 13-acetate ( PMA ), interleukin-1 (IL-1), IL-3, IL-6, granulocyte-macrophage colony stimulating factor ( GM-CSF ), thrombopoietin (TPO), and tumor necrosis factor-alpha (TNF-alpha) enhanced PGI2-R mRNA expression