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CCL4 — NFKB1
Pathways - manually collected, often from reviews:
Text-mined interactions from Literome
Dejardin et al., Immunity 2002
:
IKKbeta dependent activation of canonical
NF-kappaB is
required for the expression but not processing of p100 and for the expression of proinflammatory molecules including VCAM-1,
MIP-1beta , and MIP-2 in response to LTbetaR ligation
Wong et al., Allergy 2006
:
The induction of RANTES by SCF or TNF-alpha was mediated by ERK and
NF-kappaB , respectively, and SCF
induced MIP-1beta release was mediated by ERK
Reyes-Gordillo et al., Biochim Biophys Acta 2007
(Carbon Tetrachloride Poisoning...) :
Administration of CCl4 induced the translocation of NF-kappaB to the nucleus ;
CCl4 induced
NF-kappaB DNA binding activity was blocked by curcumin treatment
Chávez et al., J Appl Toxicol 2008
(Liver Cirrhosis, Experimental) :
Resveratrol prevents fibrosis,
NF-kappaB activation and TGF-beta increases
induced by chronic
CCl4 treatment in rats
Lakshmanan et al., J Immunol 2007
:
Caspase-4 interacts with TNF receptor associated factor 6 and mediates lipopolysaccharide induced
NF-kappaB dependent production of IL-8 and CC
chemokine ligand 4 ( macrophage-inflammatory protein-1 ) ... Because a specific
NF-kappaB inhibitor
blocked LPS induced IL-8 and
CCL4 mRNA expression as well as IL-8 and CCL4 secretion in THP1 cells, we investigated the role of caspase-4 in NF-kappaB signaling