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RAD51 — STAT5A
Protein-Protein interactions - manually collected from original source literature:
Studies that report less than 10 interactions are marked with *
Text-mined interactions from Literome
Slupianek et al., Mol Cell Biol 2002
(Cell Transformation, Neoplastic...) :
We show that
STAT5 is
essential for FTK dependent upregulation of
RAD51 , which plays a central role in homology dependent recombinational repair ( HRR ) of DNA double-strand breaks ( DSBs )
Turner et al., Leukemia 2006
(Cell Transformation, Neoplastic) :
Following transformation, FTKs induce resistance to genotoxic attack by upregulating DNA repair mechanisms such as
STAT5 dependent
RAD51 transcription